| bioRxiv
New research studies from our lab link SMAD2 genetic mutations associated with congenital heart disease to defective kidney tissue development, patterning, and function
Led by our most recent Ph.D. graduate, Rohan Bhattacharya, we discovered that mutations in the SMAD2 gene, typically associated with congenital heart disease, also lead to defective kidney tissue development, patterning, and function.